Search results for "Cortical Vein"

showing 4 items of 4 documents

Microcirculation after cerebral venous occlusions as assessed by laser Doppler scanning.

1997

✓ Research on cerebral venous circulation disturbances (CVCDs) has been limited partly by the paucity of animal models that produce consistent venous infarction. Occlusion of two adjacent cortical veins in rats by means of a photochemical thrombotic technique provides a minimally invasive, clinically relevant, and reproducible model suited to study the pathophysiology of CVCDs. In this study, the effects of venous occlusion on regional cortical blood flow and the brain damage that ensues were evaluated. Cortical vein occlusion was induced by photoactivation of rose bengal via 100-µm fiberoptic illumination. The cerebral venous flow pattern was examined using fluorescence angiography until 9…

Cerebral veinsMalemedicine.medical_specialtyBrain IschemiaInternal medicineOcclusionLaser-Doppler FlowmetryMedicineAnimalsRats Wistarbusiness.industryCerebral infarctionMicrocirculationBlood flowAnatomyLaser Doppler velocimetrymedicine.diseaseCortical VeinCerebral VeinsVein occlusionRatsDisease Models AnimalCerebral blood flowCerebrovascular CirculationCardiologybusinessJournal of neurosurgery
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Local Cerebral Blood Flow in a Rat Cortical Vein Occlusion Model

1996

The symptoms following sinus and vein occlusion observed in patients and experimental animals display a considerable variability that so far remains largely unexplained. In a rat cortical vein occlusion model using a photochemical thrombotic technique, we examined changes in the cerebral venous flow pattern by fluorescence angiography and regional cerebral blood flow (rCBF) and cerebral blood volume fraction (CBVF) by a modern laser Doppler “scanning” technique. Brain damage was assessed histologically. Fluorescence angiographic findings fell into two groups: group A, rats with an altered venous flow pattern after occlusion (n = 12), and group B, rats with interruption of blood flow and/or…

MaleCerebral veinsPathologymedicine.medical_specialtyIschemia030218 nuclear medicine & medical imaging03 medical and health sciencesCerebral circulation0302 clinical medicineOcclusionLaser-Doppler FlowmetryAnimalsMedicineFluorescein AngiographyRats WistarCerebral perfusion pressureCerebral CortexBlood Volumebusiness.industryIntracranial Embolism and Thrombosismedicine.diseaseCortical VeinVein occlusionRatsNeurologyCerebral blood flowCerebrovascular CirculationNeurology (clinical)Cardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgeryJournal of Cerebral Blood Flow & Metabolism
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Novel complement C1 inhibitor BSF468248 does not improve brain damage after cortical vein occlusion

2003

BSF468248 is a novel potent complement C1 inhibitor. To determine whether BSF468248 is effective against focal cerebral ischemia, we evaluated the change of cerebral blood flow (CBF) and infarction volume using a photochemically-induced cortical vein occlusion model in rats in blind studies. In 22 Wistar rats, two adjacent cortical veins were occluded by photochemical thrombosis and fiberoptic illumination under controlled anesthesia and ventilation. Just after the occlusion, BSF468248 or physiological saline was administrated. In the low-dose study, a treatment group (n = 7) was administered BSF468248 1 mg/kg bolus and 1 mg/kg continuously for 30 min. The same volume of saline was given to…

MaleCerebral veinsPhotochemistrymedicine.medical_treatmentComplement C1 Inactivator ProteinsRats Sprague-DawleyBolus (medicine)OcclusionmedicineAnimalsRats WistarInfusions IntravenousSalineCerebral Cortexbusiness.industryCerebral InfarctionBlood flowCortical Veinmedicine.diseaseCerebral VeinsThrombosisRatsDisease Models AnimalTreatment OutcomeCerebral blood flowRegional Blood FlowBrain InjuriesCerebrovascular CirculationAnesthesiaIntracranial ThrombosisbusinessOligopeptidesMethods and Findings in Experimental and Clinical Pharmacology
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C1-esterase inhibitor reduces infarct volume after cortical vein occlusion.

1999

In order to clarify the role of complement as a mediator of cerebral infarct growth, we inhibited the classical complement activation pathway in a photochemical cortical vein occlusion model. Immediately after occlusion, rats were infused with either 0.9% saline (vehicle), or C1-esterase inhibitor (C1-INH) over 30 min. Regional cerebral blood flow (rCBF) decreased after occlusion, and was about 50% of baseline after 2 h. No difference was noted between experimental groups. Mean arterial blood pressure (MABP) and arterial blood gases were likewise unaffected by the treatment. However, administration of C1-INH had significantly reduced infarct volume by 72%, as evaluated after 5 days survival…

MalePhotochemistryHemodynamicsComplement C1 Inactivator ProteinsVeinsOcclusionmedicineAnimalsCerebral perfusion pressureRats WistarMolecular BiologyCerebral infarctionbusiness.industryGeneral NeuroscienceCerebral Infarctionmedicine.diseaseCortical VeinRatsBlood pressureCerebral blood flowAnesthesiaCerebrovascular CirculationArterial bloodNeurology (clinical)businessDevelopmental BiologyBrain research
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